Diabetes, Heart and Cardiovascular Diseases News Chronicle.  Diabetes, Cardiovascular and Heart Diseases
 Article 196
    Published on May 25, 2017

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Longer Telomere Length And Anti-aging Benefits With Physical Exercise And Workouts

Researchers at Brigham Young University, Provo, Utah, United States have found that an individual can slowdown the biological aging process by almost ten years with vigorous physical exercise when compared with an individual who is with sedentary lifestyle or with couch potato behavior. The researchers conducted survey among those individuals who participated in the National Health and Nutrition Examination Survey, United States between 1999 and 2002. Total number of participants were 5,823 and they collected details from them related to their lifestyle, exercise details and demographic information (such as their education, nationality, religion and ethnicity etc). They also measured their telomere length.


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Researchers compared telomere length and exercise levels of the participants. Results shows marked differences in the telomere length between individuals who do regular physical exercise and in those individuals with sedentary lifestyle or with couch potato behavior. Researchers found that when compared with individuals with sedentary lifestyle or no physical activity, individuals who are with high levels of physical activity were biologically younger by nine years and individuals who are with moderate amount of exercise were biologically younger by seven years.


Longer telomere length and biological anti-aging benefits with physical exercise and workouts.
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Researchers says regular physically active individuals can have longer telomere length and less biological aging but they don't know the reason for longer telomere length with regular physical exercise. Author of the study was Professor Larry Tucker, from the Brigham Young University and the study findings were published in April issue of the journal Preventative Medicine.


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Breast Milk And Omega-3 Linked To Lower Risk Of Type 1 Diabetes

A study by researchers from the National Institute of Health and Welfare, Helsinki, Finland shows a link between lower risk of the development of type 1 diabetes (T1D) related auto-immunity and the breastfeeding of omega-3 polyunsaturated fatty acid to new born babies.


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Researchers found that risk of type 1 diabetes related to auto-immunity reduces as quantity of breast milk increased. But risk of type 1 diabetes related to auto-immunity increases as the cow's milk-based formula quantity increases. Researchers says type 1 diabetes (T1D) associated auto-immunity is related to status of fatty acid during infancy and particularly omega 3 fatty acids and they are associated with reduced risk of type 1 diabetes (T1D).

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To find out the association between omega-3 fatty acids and type 1 diabetes (T1D) development, they studied 7,782 born babies aged between 3 and 24 months who are at genetic risk of the development of type 1 diabetes (T1D). Blood samples were collected annually up to age of 15 years and they checked islet cell or pancreas beta cell auto-antibodies. They also collected details about mother's breastfed milk and formula (through which infants receive fatty acids).


Type 1 diabetes insulin auto-immunity with breastfeeding of omega 3 polyunsaturated fatty acid.
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Their study results shows higher levels of omega-3 fatty acids were associated with lower risk of early insulin auto-immunity. They also found that infant's fatty acid levels reflects type of milk feeding. They say infant can get protective mechanism against type 1 diabetes (T1D) with omega-3 fatty acids consumption. It is important that mother should consume omega-3 fatty acids during breastfeeding. Researchers says further studies are required. Author of the study was Dr Sari Niinista (Niinisto) and the study findings were published in the journal Diabetologia.


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