Diabetes, Heart and Cardiovascular Diseases News Chronicle Diabetes, Cardiovascular and Heart Diseases. Article 60
Published on September 18, 2016 at 07:30 PM


Role Of Pioglitazone Diabetes Drug In Preventing Breast Cancer

Scientists discovered NAF-1 protein in our body which is associated with growth of breast cancer. Type 2 diabetes (T2D) drug pioglitazone helped in bringing down NAF-1 protein levels there by preventing proliferation of cancer cells and stopping tumors. Scientists says NAF-1 protein is associated with other cancers such as prostate, cervical, gastric, liver, and laryngeal cancer. Scientists says improving pioglitazone drug to address NAF-1 protein can bring new weapon to fight against cancer. Coauthor of the study is Prof Rachel Nechushtai, Hebrew University of Jerusalem.

       
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Pioglitazone : Pioglitazone is a thiazolidinedione class prescription drug to treat type 2 diabetes (T2D). The drug decreases blood glucose levels and decreases insulin resistance so that cells can take more glucose.

 

Risks With Over Eating Of Omega-3 Fatty Acids

A study done by Dr Guy Fagherazzi, France says that over consumption of oily fish such as salmon, mackerel or sardines increases risk to type ii diabetes (T2D) by 26 percent. Researchers studied 71,334 women for 14 years who ate maximum oily fish food and came to conclusion. Researchers says it is not good to consume omega-3 foods greater than recommended daily nutritional requirements. Dr Guy Fagherazzi, of University Paris-Saclay, Villejuif, France is a researcher in this study and the study findings were presented at the European Association for the Study of Diabetes (EASD) meeting in Munich, Germany.

       
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Omega-3 : Omega 3 fatty acids are polyunsaturated fatty acids (PUFAs) and important for our body metabolism. Nutritionists around the world recommends eating omega-3 foods to prevent heart diseases, depression, boosts brain power, strengthen bones, cancer, dementia etc.

 

Treatment To Diabetic Neuropathy

25 percent of diabetes patients suffer with nerves pains in their feet. Researchers from Northwestern University, conducted experiments with mice for the treatment of diabetic neuropathy. They says diabetic neuropathy can be prevented and reversed by depleting a GM3 chemical through genetic modifications. GM3 synthase is an enzyme that makes GM3. Researchers are using gene therapy ointment to footpad of diabetic mice to remove GM3 and GM3 synthase to treat the existing neuropathy. Researchers says that problem is same for both mice and humans and the same can be used for the treatment of diabetic neuropathy in humans. First author of the study is Dr Daniela Menichella, Assistant Professor of Neurology (Neuromuscular Disease), Northwestern Memorial Hospital (includes Prentice Women's Hospital), a Feinberg affiliated hospital. The study results were published in Molecular Pain.

       
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Diabetic neuropathy : Chronically high levels of glucose in blood can damages small blood vessels. How this happens is not yet clear. Damaged blood vessels prevent supply of oxygen and glucose to nerves and prevents its functionality and this is called diabetic neuropathy. Diabetic neuropathy patients gets pain and loss of feeling in the hands, feet, arms and legs. One can prevent or stop progress of diabetic neuropathy by controlling high blood sugar levels.

Nerves : Nerves send and receive messages between body organs and brain by electrical impulses or signals. Nerves are of three types.

 

SGLT2 Inhibitor Drug Ertugliflozin

Merck in partnership with Pfizer Inc. announced that their phase 3 (VERTIS SITA2) study of oral drug ertugliflozin (SGLT2 inhibitor) for the treatment of type 2 diabetes (T2D) met the trial end point. Ertugliflozin 5 mg and 15 mg daily doses showed significant greater reduction of A1C. The study results were presented during 52nd Annual Meeting of the European Association for the Study of Diabetes (EASD) in Munich, Germany. Merck and Pfizer are planning to submit applications to U.S. FDA for the approval of three drugs by the end of 2016. They are

       
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